Author(s): Olajide OJ; La Rue C; Bergdahl A; Chapman CA;
Increasing evidence suggests that mitochondrial dysfunction and aberrant release of mitochondrial reactive oxygen species (ROS) play crucial roles in early synaptic perturbations and neuropathology that drive memory deficits in Alzheimer's disease (AD). ...
Article GUID: 36275011
Author(s): Batallán Burrowes AA; Sundarakrishnan A; Bouhour C; Chapman CA;
Activation of estrogen receptors is thought to modulate cognitive function in the hippocampus, prefrontal cortex, and striatum by affecting both excitatory and inhibitory synaptic transmission. The entorhinal cortex is a major source of cortical sensory and ...
Article GUID: 34399010
Author(s): Olajide OJ; Chapman CA;
The hippocampus and entorhinal cortex (EC) accumulate amyloid beta peptides (Aß) that promote neuropathology in Alzheimer's disease, but the early effects of Aß on excitatory synaptic transmission in the EC have not been well characterized. To assess th ...
Article GUID: 34144329
Author(s): Olajide OJ, Suvanto ME, Chapman CA
The entorhinal cortex (EC) is a vital component of the medial temporal lobe, and its contributions to cognitive processes and memory formation are supported through its extensive interconnections with the hippocampal formation. During the pathogenesis of Al ...
Article GUID: 33495355
Author(s): Sparks DW, Chapman CA
J Neurophysiol. 2016 08 01;116(2):658-70 Authors: Sparks DW, Chapman CA
Article GUID: 27146979
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