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TMS and H1-MRS measures of excitation and inhibition following lorazepam administration.

Author(s): Ferland MC, Therrien-Blanchet JM, Proulx S, Klees-Themens G, Bacon BA, Vu TTD, Théoret H

This study aimed at better understanding the neurochemistry underlying TMS and MRS measurements as it pertains to GABAergic activity following administration of allosteric GABAA receptor agonist lorazepam. Seventeen healthy adults (8 females, 26.0 ± 5.4 yea...

Article GUID: 33246064
Prefrontal Cortex and Multiparity in Lactation.

Author(s): Opala EA, Verlezza S, Long H, Rusu D, Woodside B, Walker CD

Neuroscience. 2019 Aug 19;: Authors: Opala EA, Verlezza S, Long H, Rusu D, Woodside B, Walker CD

Article GUID: 31437474
17β-Estradiol infusions into the dorsal striatum rapidly increase dorsal striatal dopamine release in vivo.

Author(s): Shams WM, Sanio C, Quinlan MG, Brake WG

Neuroscience. 2016 08 25;330:162-70 Authors: Shams WM, Sanio C, Quinlan MG, Brake WG

Article GUID: 27256507
Effect of electrolytic lesions of the dorsal diencephalic conduction system on the distribution of Fos-like immunoreactivity induced by rewarding electrical stimulation.

Author(s): Fakhoury M, Voyer D, Lévesque D, Rompré PP

Neuroscience. 2016 Oct 15;334:214-225 Authors: Fakhoury M, Voyer D, Lévesque D, Rompré PP

Article GUID: 27514573
Dopamine Signaling Is Critical for Supporting Cue-Driven Behavioral Control.

Author(s): Iordanova MD

Neuroscience. 2019 May 17;: Authors: Iordanova MD

Article GUID: 31103706
Dopaminergic enhancement of excitatory synaptic transmission in layer II entorhinal neurons is dependent on D₁-like receptor-mediated signaling.

Author(s): Glovaci I, Caruana DA, Chapman CA

Neuroscience. 2014 Jan 31;258:74-83 Authors: Glovaci I, Caruana DA, Chapman CA

Article GUID: 24220689
Serotonin 5-HT1A Receptor-Mediated Reduction of Excitatory Synaptic Transmission in Layers II/III of the Parasubiculum.

Author(s): Carter F, Chapman CA

Neuroscience. 2019 May 15;406:325-332 Authors: Carter F, Chapman CA

Article GUID: 30902681
Title:Effect of electrolytic lesions of the dorsal diencephalic conduction system on the distribution of Fos-like immunoreactivity induced by rewarding electrical stimulation.
Authors:Fakhoury M,  Voyer D,  Lévesque D,  Rompré PP
Link:https://www.ncbi.nlm.nih.gov/pubmed/27514573?dopt=Abstract
DOI:10.1016/j.neuroscience.2016.08.002
Category:Neuroscience
PMID:27514573
Dept Affiliation: CSBN
1 Department of Neurosciences, Faculty of Medicine, Université de Montréal, Montreal, Quebec, Canada.
2 Faculty of Pharmacy and FRQ-S Research Group on Central Nervous System, Université de Montréal, Montreal, Quebec, Canada.
3 Department of Neurosciences, Faculty of Medicine, Université de Montréal, Montreal, Quebec, Canada; FRQ-S Research Group in Behavioral Neurobiology, Concordia University, Montreal, Quebec, Canada. Electronic address: pierre-paul.rompre@umontreal.ca.

Description:

Effect of electrolytic lesions of the dorsal diencephalic conduction system on the distribution of Fos-like immunoreactivity induced by rewarding electrical stimulation.

Neuroscience. 2016 Oct 15;334:214-225

Authors: Fakhoury M, Voyer D, Lévesque D, Rompré PP

Abstract

The dorsal diencephalic conduction system (DDC) is an important pathway of the brain reward circuitry, linking together forebrain and midbrain structures. The present work was aimed at describing the effect of a DDC lesion on the distribution of Fos-like immunoreactivity (FLIR) following intracranial self-stimulation (ICSS) of the lateral hypothalamus (LH). Rats were implanted with monopolar electrodes and divided into three groups; the first two groups were trained to self-stimulate at the LH, whereas the third group received no stimulation and served as a control. Among the two groups that were trained for ICSS, one of them received a lesion at the DDC and was tested for ICSS on the subsequent 5days. On the last day of testing, control rats were placed in operant chambers without receiving any stimulation, and the remaining rats were allowed to receive the stimulation for 1h. All rats were then processed for FLIR. As previously shown, a lesion at the DDC resulted in significant attenuations of the rewarding effectiveness of LH stimulation. Results also show a higher FLIR in several reward-related areas following LH stimulation, especially in the hemisphere ipsilateral to the stimulation electrode. Compared to non-lesioned rats, lesioned animals had lower FLIR in certain brain regions, suggesting that those regions that were activated by the rewarding stimulation may be functionally interconnected with the DDC.

PMID: 27514573 [PubMed - indexed for MEDLINE]